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Status Epilepticus

Continuous seizure activity >= 5 min, or >= 2 seizures without full recovery; emergency IV Lorazepam (0.1 mg/kg) then IV Levetiracetam/Fosphenytoin.

Focal vs. Generalized

Focal (originates in 1 hemisphere, may be aware or impaired awareness); Generalized (simultaneous bilateral hemispheres, e.g., Absence, Tonic-Clonic).

Myasthenia Gravis

Antibodies to post-synaptic ACh receptors; fatigable weakness worsens with use; thymoma association; treat with Pyridostigmine.

Lambert-Eaton

Antibodies to pre-synaptic P/Q-type calcium channels; weakness improves with repetitive use; Small Cell Lung Cancer association.

Stepwise Status Epilepticus Emergency Resuscitation Protocol

Status epilepticus leads to permanent neuronal injury and systemic acidosis if seizure duration exceeds 30 minutes. The resuscitation protocol is structured into time-critical phases:

0–5 Minutes: Stabilization Phase

Airway, breathing, circulation. Check bedside capillary fingerstick blood glucose immediately. Administer IV dextrose + thiamine if hypoglycemic. Obtain IV access.

5–20 Minutes: First-Line Therapy (Benzodiazepines)

IV Lorazepam (Ativan): 4 mg IV over 2 min (repeat once at 5-10 min). If IV access unavailable: IM Midazolam 10 mg (or rectal diazepam).

20–40 Minutes: Second-Line Therapy (Urgent Control)

Administer non-sedating IV antiepileptic: IV Levetiracetam (Keppra) 60 mg/kg (max 4500 mg), IV Fosphenytoin 20 mg PE/kg, or IV Valproate sodium 40 mg/kg.

40+ Minutes: Refractory Status Epilepticus

Endotracheal intubation and continuous general anesthetic infusion: Propofol, Midazolam, or Ketamine with continuous bedside EEG monitoring for burst suppression.

Neuromuscular Junction Disorders: Myasthenia Gravis vs. Lambert-Eaton

FeatureMyasthenia Gravis (MG)Lambert-Eaton Myasthenic Syndrome (LEMS)
Target AntigenPost-synaptic Nicotinic Acetylcholine Receptors (AChR) or MuSKPre-synaptic Voltage-Gated P/Q-type Calcium Channels (VGCC)
Effect of Repetitive UseMuscle weakness worsens with use (fatigability)Muscle strength improves with exercise (facilitation)
Ocular InvolvementPtosis and diplopia common initial presentation (> 50%)Ocular involvement rare; proximal limb weakness dominates
Autonomic SymptomsAbsentPresent: Dry mouth, erectile dysfunction, constipation
Deep Tendon ReflexesNormalDecreased or absent (may return after brief exercise)
Underlying NeoplasmThymoma (15%) or thymic hyperplasia (65%); chest CT mandatorySmall Cell Lung Carcinoma (SCLC) (> 50%)
OMM Board Correlate: Post-Ictal Care & Cranial Strain
  • Post-Ictal Hypertonicity: Tonic-clonic seizures cause profound lactic acidosis and generalized myofascial hypertonicity. Gentle indirect myofascial release and rib raising aid in respiratory recovery.
  • Cranial RTM Strains: Compression of the sphenobasilar synchondrosis (SBS) is common following head trauma associated with falls during seizure episodes. The CV4 technique and venous sinus drainage encourage normal CRI fluctuation.
  • Contraindication: Never perform HVLA thrusting techniques in patients with active or uncontrolled seizure disorders.
Board Traps & Common Distractors
  • In myasthenic crisis or Guillain-Barré syndrome, never rely on pulse oximetry or arterial blood gas (ABG) to detect respiratory failure; pulse oximetry remains normal until terminal arrest. Measure Forced Vital Capacity (FVC) and Negative Inspiratory Force (NIF); intubate if FVC < 20 mL/kg or NIF < -30 cm H2O.
  • Avoid aminoglycosides, fluoroquinolones, macrolides, beta-blockers, and magnesium in patients with Myasthenia Gravis; all severely impair neuromuscular transmission and can precipitate fatal myasthenic crisis.
  • In patients presenting with new-onset status epilepticus, always check bedside glucose; hypoglycemia is rapidly fatal if untreated, whereas administering phenytoin alone will not terminate hypoglycemic convulsions.