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Resuscitation Quick Ribbon (First 2 Minutes)

High-Acuity
SJS / TEN Burn Transfer:TEN (> 30% BSA) or SJS/TEN overlap (10–30% BSA) mandates immediate Burn ICU transfer
Positive Nikolsky Sign:Gentle lateral tangential pressure on normal-appearing skin sloughs epidermis (acantholysis)
Culprit Drug Cessation:Immediately discontinue all high-risk drugs: Allopurinol, Antiepileptics, Sulfonamides, NSAIDs
DRESS Syndrome Steroids:Systemic Methylprednisolone 1–2 mg/kg/day IV for severe visceral organ involvement (hepatitis/nephritis)
Purpura Fulminans Timing:Meningococcemia: Administer Ceftriaxone 2g IV + Vancomycin immediately; do not delay for LP

Bottom-Line Clinical Pearl

Life-threatening dermatologic emergencies present with systemic toxicity and mucosal involvement. In Stevens-Johnson Syndrome (SJS) and Toxic Epidermal Necrolysis (TEN), drug-induced keratinocyte apoptosis causes extensive epidermal detachment. Nikolsky sign is positive (gentle lateral pressure sloughs skin). Involve the burn center early for TEN (> 30% BSA involvement) or SJS/TEN overlap (10–30% BSA); immediately discontinue all potential culprit medications (allopurinol, antiepileptics, sulfonamides, NSAIDs). DRESS syndrome presents with facial edema, widespread morbilliform rash, fever, and eosinophilia with visceral involvement (hepatitis). In meningococcemia, purpura fulminans progresses in hours; administer IV Ceftriaxone immediately.

1. Stevens-Johnson Syndrome (SJS) & Toxic Epidermal Necrolysis (TEN)

SJS and TEN are severe, immune-mediated mucocutaneous adverse drug reactions characterized by extensive Fas/Fas-ligand and granulysin-mediated keratinocyte apoptosis. They represent a spectrum of disease defined by the percentage of total body surface area (BSA) with epidermal detachment:

ClassificationEpidermal Detachment (% BSA)Mucosal InvolvementMortality Rate & Disposition
Stevens-Johnson Syndrome (SJS)< 10% BSA detachment>= 2 mucosal surfaces involved (eyes, oral cavity, genitalia) in > 90%1–5% mortality. Inpatient telemetry or specialized dermatology unit.
SJS / TEN Overlap10% to 30% BSA detachmentBilateral conjunctivitis, painful oral erosions, hemorrhagic crusting10–15% mortality. Transfer to regional Burn ICU.
Toxic Epidermal Necrolysis (TEN)> 30% BSA detachment (often 50–80%)Severe multi-mucosal sloughing, respiratory tract detachment, intestinal shedding30–50% mortality. MANDATORY IMMEDIATE BURN CENTER TRANSFER.
SCORTEN Parameter (Assess at 24h of presentation)Risk Factor ThresholdScore Point Assigned
1. AgeAge >= 40 years1 Point
2. Heart RateTachycardia >= 120 beats/minute1 Point
3. Associated MalignancyKnown active cancer / hematologic malignancy1 Point
4. Detached / Detachable BSAEpidermal detachment > 10% BSA at day 11 Point
5. Serum Urea (BUN)BUN > 28 mg/dL (10 mmol/L)1 Point
6. Serum GlucoseBlood glucose > 252 mg/dL (14 mmol/L)1 Point
7. Serum BicarbonateBicarbonate < 20 mEq/L (acidosis)1 Point
Mortality Risk by SCORTENScore 0–1: 3.2%; Score 2: 12.1%; Score 3: 35.3%; Score 4: 58.3%; Score >= 5: 90.0%Calculates ICU/Burn mortality

2. DRESS Syndrome (Drug Reaction with Eosinophilia & Systemic Symptoms)

DRESS syndrome is a delayed, severe hypersensitivity reaction typically developing 2 to 8 weeks after starting a culprit medication (most commonly Carbamazepine, Phenytoin, Lamotrigine, Allopurinol, or Sulfasalazine). It is characterized by the hallmark clinical triad: 1) Extensive morbilliform rash with facial edema; 2) Fever (> 38.5°C); 3) Internal organ involvement (hepatitis with elevated ALT in 80%, acute interstitial nephritis in 30%, myocarditis):

  • Facial Edema Hallmark: Marked, symmetrical periorbital and facial edema is present in > 75% of patients with DRESS, distinguishing it from simple drug exanthems.
  • Laboratory Confirmation: Absolute eosinophilia (> 700/uL or > 10% of differential) and atypical lymphocytes on peripheral blood smear.
  • Emergency Treatment: 1) Immediately stop the culprit medication; 2) Administer systemic corticosteroids (Methylprednisolone 1–2 mg/kg/day IV for severe organ involvement, transitioned to oral prednisone tapered over 2 to 3 months to prevent relapses); 3) Hospital admission to monitored telemetry bed.

3. Purpura Fulminans & Meningococcemia

Meningococcemia (Neisseria meningitidis bacteremia) produces endotoxin-mediated endothelial damage, microvascular thrombosis, and acute Disseminated Intravascular Coagulation (DIC). Petechiae rapidly coalesce into painful, geographic, purple-black necrotic purpura (Purpura Fulminans) accompanied by Waterhouse-Friderichsen syndrome (bilateral adrenal hemorrhage):

Critical Pitfall / Contraindication

Meningococcemia Antibiotic Rule: In a febrile patient with petechiae or rapidly spreading purpura, administer IV Ceftriaxone (2g IV) immediately. DO NOT DELAY ANTIBIOTICS for a lumbar puncture or CT scan. Seconds matter: patients can progress from initial fever to irreversible septic shock and limb necrosis in under 4 hours.

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